New mechanism of anti-hepatitis B virus infection

After more than 4 years of unremitting efforts, the research team led by Professor Xiong Sidong at the Institute of Immunobiology of Fudan University has found that the TRIM22 molecule in the human body is important in the prevention of hepatitis B virus infection in the study of the inherent immune mechanism of hepatitis B virus infection The results of this research have been published in the latest issue of the internationally renowned journal "Journal of Hepatology".

The scientific research team has confirmed through numerous in vitro and in vivo experiments that the TRIM22 molecule is of great significance for inhibiting the replication of hepatitis B virus and preventing the occurrence of hepatitis B. In further research, it was found that TRIM22 can significantly inhibit the activity of the hepatitis B virus core promoter, thereby achieving the purpose of inhibiting virus replication. This achievement is to further study the interaction of hepatitis B virus and the body's immune system, and to further clarify the clinical The new mechanism of action of commonly used antiviral drugs will have a major and far-reaching impact, and will also provide new targets and research ideas for the design and development of new generation anti-hepatitis B virus drugs.

Recent researches by scientists have found that a TRIM22 molecule generally exists in the human body, but people do not understand its role and function. Research by the team led by Xiong Sidong shows that this new molecule is actually an inherent immune molecule that is beneficial to the human body. After some people are infected with hepatitis B virus, they can quickly stimulate the body's innate immunity and specific immune mechanism to inhibit viral replication, clear the hepatitis B virus, and then heal. Among them, TRIM22 molecule plays an important role; while others infected with hepatitis B virus, because of TRIM22 molecule Inability to play a timely and effective role leads to hepatitis B virus infection, and will further develop into chronic hepatitis and even liver cancer.

In addition, interferon is currently one of the effective drugs for the treatment of chronic hepatitis B. Related studies have found that interferon may inhibit the replication of hepatitis B virus by stimulating the TRIM22 molecule. The research results reveal for the first time the important inherent immune mechanism of the human body against hepatitis B virus infection, which is of great significance to the scientific community to continue to develop a new generation of drugs against hepatitis B virus infection

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